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Samant P.S.,Sardar Patel Post Graduade Institute of Dental and Medical science | Tewari T.,Uttar Pradesh Dental College and Research Center | Tondon A.,Uttar Pradesh Dental College and Research Center | Chandra R.,Career Dental College
Indian Journal of Public Health Research and Development | Year: 2012

Endodontic management of immature non vital permanent teeth in young pediatric patients is a great challenge to dentists. The walls of the root canals are frequently divergent and open apexes make debridement and obturation difficult. Thus closure of root apex is very essential for success of the endodontic treatment. Although different materials are used for the apexification procedure, calcium hydroxide is the material of choice for apical barrier formation and healing. There are different opinions regarding frequency of CaOH dressing change to induce complete closure of the apex. Literature suggests that dressing should be changed frequently. Therefore the aim of the present article is to report the successful closure of root apex in pulpless permanent incisors with wide open apices in a pediatric patients using CaOH dressing. Source

Roopashree M.R.,Uttar Pradesh Dental College and Research Center | Gondhalekar R.V.,Uttar Pradesh Dental College and Research Center | Shashikanth M.C.,Uttar Pradesh Dental College and Research Center | George J.,Uttar Pradesh Dental College and Research Center | And 2 more authors.
Journal of Oral Pathology and Medicine | Year: 2010

Oral lichen planus (OLP) is a T-cell-mediated chronic inflammatory oral mucosal disease of unknown etiology. OLP presents as white striations, white papules, white plaques, erythema, erosions, or blisters affecting predominantly the buccal mucosa, tongue and gingiva. Both antigen-specific and non-specific mechanisms are hypothesized to be involved in the pathogenesis of oral lichen planus (OLP). Antigen-specific mechanisms in OLP include antigen presentation by basal keratinocytes and antigen-specific keratinocyte killing by CD8+ cytotoxic T cells. Non-specific mechanisms include mast cell degranulation and matrix metalloproteinase activation in OLP lesions. These mechanisms may combine to cause T cell accumulation in the superficial lamina propria, basement membrane disruption, intra-epithelial T cell migration and keratinocyte apoptosis in OLP. The various hypotheses proposed for pathogenesis of oral lichen planus are discussed in this review. © 2010 John Wiley & Sons A/S. Source

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