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Dihok, Iraq

Ali I.,University of Dohuk | Moiron S.,VMware | Fleury M.,University of Essex | Ghanbari M.,University of Essex | Ghanbari M.,Sharif University of Technology
IEEE Wireless Communications | Year: 2013

Multiple description video coding can exploit path diversity to improve error resiliency by generating several independently decodable video descriptions and sending them over different paths. Such paths are readily available in ad hoc wireless networks. To approach the original quality, all descriptions need to be successfully received. However, in a wireless channel, error bursts or link disruption may still affect one or more descriptions, resulting in significant drops in video quality. This article describes a lower complexity yet effective scheme for video streaming over such wireless networks. In order to improve error resiliency, the scheme combines unequal packet importance with the advantages of path diversity. The scheme can be implemented with a minimal increase in computational complexity, as spatial decomposition of video frames is employed to generate the descriptions. Experimental results for an ad hoc wireless network show that a significant objective quality gain can be achieved (up to 3 dB) in the presence of signal shadow fading. © 2013 IEEE. Source

Sheet M.H.,University of Dohuk
ARPN Journal of Engineering and Applied Sciences | Year: 2012

This paper focused on studying the effect of silt quantity in soils on the performance of synthetic fabrics called Geotextiles, which is a standard set by the U.S. Army Corps of Engineers to evaluate the degree of fabric clogging. Certain hydraulic relationships were established to determine the effect of silt quantity in soils on the performance of Geotextiles. From the results we found out a relation between silt quantity and gradient ratio, so the increase of silt quantity in soils leads to increase gradient ratio and that causes increase clogging degree of the synthetic fabric (Geotextile). © 2006-2012 Asian Research Publishing Network (ARPN). Source

Al-Allawi N.A.,University of Dohuk
Annals of Saudi medicine | Year: 2010

Hemoglobin Barts hydrops fetalis syndrome is the most severe and generally fatal clinical phenotype of alpha-thalassemia. We diagnosed a fetus at 23-weeks gestation with having hydrops fetalis, by ultrasound. At 32 weeks, intrauterine death was detected. Molecular studies revealed that the fetus had the hemoglobin Barts hydrops fetalis syndrome due to homozygosity for the Mediterranean alpha-thalassemia deletion. This clinical phenotype is generally rare in the Eastern Mediterranean, and this is the first report of this syndrome from Iraq. Techniques for molecular characterization became available only very recently in this country, in a diagnostic setting. Thus, the detection of further cases might be expected in future. Source

Hamamy H.,University of Geneva | Makrythanasis P.,University of Geneva | Al-Allawi N.,University of Dohuk | Muhsin A.A.,Hematology Oncology Unit | Antonarakis S.E.,University of Geneva
BMC Medical Genetics | Year: 2014

Background: Inherited thrombocytopenias (IT) are a heterogeneous group of rare diseases characterized by a reduced number of blood platelets. The frequency of IT is probably underestimated because of diagnostic difficulties and because not all the existing forms have as yet been identified, with some patients remaining without a definitive diagnosis. Exome Sequencing has made possible the identification of almost all variants in the coding regions of protein-coding genes, thereby providing the opportunity to identify the disease causing gene in a number of patients with indefinite diagnoses, specifically in consanguineous families. Case presentation: Familial thrombocytopenia with small size platelets was present in several members of a highly consanguineous family from Northern Iraq. Genotyping of all affected, their unaffected siblings and parents, followed by exome sequencing revealed a strong candidate loss of function variant in a homozygous state: a frameshift mutation in the gene. The protein encoded by this gene is known to be a cytosolic adaptor molecule expressed by T, natural killer (NK), myeloid cells and platelets, and is involved in platelet activation and controls the expression of interleukin-2. Knock-out mice were reported to show isolated thrombocytopenia. Conclusion: Inherited thrombocytopenias differ in their presentation, associated features, and molecular etiologies. An accurate diagnosis is needed to provide appropriate management as well as counseling for the individuals and their family members. Exome sequencing may become a first diagnostic tool to identify the molecular basis of undiagnosed familial IT. In this report, the clinical evaluation combined with the power and efficiency of genomic analysis defined the FYB gene as the possible underlying cause of autosomal recessive thrombocytopenia with small platelet size. This is the first report linking pathogenic variants in FYB and thrombocytopenia in humans. © Hamamy et al. Source

Al-Allawi N.A.S.,University of Dohuk | Al-Mousawi B.M.S.,University of Baghdad | Badi A.I.A.,University of Dohuk | Jalal S.D.,Sulaimaniyah University
Hemoglobin | Year: 2013

While previous studies from Iraq have focused on β-thalassemia (β-thal) mutations in the northern part of the country, inhabited mainly by Kurds, no study of significance has looked at these mutations in central or southern Iraq, which is inhabited by the Arab majority. For the latter purpose this study was initiated and 103 β-thal carriers from Baghdad at the center of the country were investigated using multiplex polymerase chain reaction (PCR) and reverse hybridization followed by sequencing. The results revealed that a total of 17 mutations were implicated, six of which, IVS-I-110 (G>A), IVS-II-1 (G>A), IVS-I-5 (G>C), codons 8/9 (+G), IVS-I-I (G>A) and codon 44 (-C), constituted 78.0% of the mutations characterized. Among the 17 mutations identified, six are reported for the first time from Iraq and include: IVS-I, 25 bp deletion, IVS-II-848 (C>A), -28 (A>C), IVS-I-130 (G>C), IVS-I-128 (T>G) and codons 41/42 (-TTCT). The findings of the current study clearly illustrate the genetic heterogeneity of the population of central Iraq, as demonstrated by the presence of a combination of Mediterranean, Asian Indian, Kurdish, Iranian, Egyptian, Saudi Arabian and Turkish mutations that is reflective of the historical background of this part of the country. Copyright © Informa Healthcare USA, Inc. Source

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