News Article | April 17, 2017
LearnHowToBecome.org, a leading resource provider for higher education and career information, has used metrics provided by the government to select the best colleges and universities in Arizona for 2017. 6 four-year schools had the qualifying scores to be included, and Arizona State University Tempe, University of Arizona and Northern Arizona University were the top three. Of the 20 two-year schools included in the ranking, Cochise College, Northland Pioneer College, Mesa Community College, GateWay Community College and Eastern Arizona College were the top five. A full list of schools is included below. “A certificate or degree can go a long way when it comes to starting or advancing a career,” said Wes Ricketts, senior vice president of LearnHowToBecome.org. “These colleges and universities in Arizona have demonstrated their value to students who want to be prepared for their role in the job market. Post-college earnings, employment resources and high program caliber were all evaluated to determine which schools belonged on our list.” To be included on Arizona’s “Best Colleges” list, schools must be regionally accredited, not-for-profit institutions. In addition to their career resources, each college is also analyzed based on additional metrics including program offerings, academic counseling, opportunities for financial aid, graduation rates and student/teacher ratios. Complete details on each college, their individual scores and the data and methodology used to determine the LearnHowToBecome.org “Best Colleges in Arizona” list, visit: The Best Four-Year Colleges in Arizona for 2017 include: Arizona Christian University Arizona State University-Tempe Northern Arizona University Ottawa University-Phoenix Prescott College University of Arizona The Best Two-Year Colleges in Arizona for 2017 include: Arizona Western College Central Arizona College Chandler-Gilbert Community College Cochise College Coconino Community College Eastern Arizona College Estrella Mountain Community College GateWay Community College Glendale Community College Mesa Community College Mohave Community College Northland Pioneer College Paradise Valley Community College Phoenix College Pima Community College Rio Salado College Scottsdale Community College South Mountain Community College Tohono O'Odham Community College Yavapai College ### About Us: LearnHowtoBecome.org was founded in 2013 to provide data and expert driven information about employment opportunities and the education needed to land the perfect career. Our materials cover a wide range of professions, industries and degree programs, and are designed for people who want to choose, change or advance their careers. We also provide helpful resources and guides that address social issues, financial aid and other special interest in higher education. Information from LearnHowtoBecome.org has proudly been featured by more than 700 educational institutions.
Parvinen K.,University of Turku |
Seppanen A.,University of Turku |
Nagy J.D.,Scottsdale Community College |
Nagy J.D.,Arizona State University
Bulletin of Mathematical Biology | Year: 2012
The question of how dispersal behavior is adaptive and how it responds to changes in selection pressure is more relevant than ever, as anthropogenic habitat alteration and climate change accelerate around the world. In metapopulation models where local populations are large, and thus local population size is measured in densities, density-dependent dispersal is expected to evolve to a single-threshold strategy, in which individuals stay in patches with local population density smaller than a threshold value and move immediately away from patches with local population density larger than the threshold. Fragmentation tends to convert continuous populations into metapopulations and also to decrease local population sizes. Therefore we analyze a metapopulation model, where each patch can support only a relatively small local population and thus experience demographic stochasticity. We investigated the evolution of density-dependent dispersal, emigration and immigration, in two scenarios: adult and natal dispersal. We show that density-dependent emigration can also evolve to a nonmonotone, "triple-threshold" strategy. This interesting phenomenon results from an interplay between the direct and indirect benefits of dispersal and the costs of dispersal. We also found that, compared to juveniles, dispersing adults may benefit more from density-dependent vs. density-independent dispersal strategies. © 2012 Society for Mathematical Biology.
News Article | October 28, 2016
Each year in the month of November, the Maricopa Community Colleges honor active duty military and veteran members of our communities with events at many of our 10 colleges. At Veteran Services Centers at each college, and the East Valley Veterans Education Center, active duty and veteran students can find academic and college success advising, career re-entry planning, and peer support. Currently, Chandler-Gilbert, South Mountain, Scottsdale, Glendale, Rio Salado and Mesa Community Colleges are designated Arizona Veteran Supportive Campuses. The following events are free and open to the public. Chandler-Gilbert Community College Thurs., Nov. 3, 1 p.m. to 4 p.m., Agave Hall Community Room and Courtyard (Sgt. Gerry Jones, USMC, at 2 p.m.) Chandler-Gilbert Community College, 2626 E Pecos Rd, Chandler The public is welcome to the 8th Annual Veterans Resource Expo, showcasing college and community resources, career, and educational opportunities for veterans. Guest speaker Sgt. Gerry Jones, USMC, renowned architect and co-founder of the Town of Carefree, will share his story of service as a Marine in World War II and Korea. Light refreshments served. GateWay to the Arts, in honor of Veterans Day and in cooperation with the GateWay Veterans Center and the United States Navy, presents “The Flying Fleet”. Join our military and color guard guests of honor as we screen this classic silent film from 1929. This event is free and open to the public. Park in west lot off of 38th Street across from the Copper Room, located in the Integration Education building, or ride the Light Rail to the GateWay Community College stop. Contact Donald Hall at hall(at)gatewaycc(dot)edu with questions. Glendale Community College honors veterans and commemorates Pearl Harbor on Nov. 10. with displays, special guest speakers, dedications, and refreshments. Veterans Day ceremonies at Estrella Mountain Community College (EMCC), in collaboration with the EMCC student Veterans Club, begin Fri., Nov. 11 with a family fun event and fitness challenge 5K run at 8 a.m., followed by ceremonies and a pancake breakfast prepared by EMCC culinary students. The festivities will include performances by EMCC dance students, patriotic displays from area veteran groups, and a formal ceremony that includes speakers from Luke Air Force Base. Proceeds from the event will directly benefit the development of The West Valley Center for Military & Veteran Success, a Veteran Success Project initiative. Breakfast is $5 per person, free for veterans and their families with military identification. Breakfast is also included as part of the registration cost for Fitness Fun Run participants. Run registration is $25 for military, veterans, and those that pre-register online through October 26. Event day registration is $35. Register by October 26 to be guaranteed a free event performance t-shirt and bag full of giveaways. Visit http://www.estrellamountain.edu/vetfunrun for more information and to register. Paradise Valley Community College hosts several events, beginning Oct. 25 with “Shout Out to Vets.” Record a quick “shout out” to thank veterans or recognize specific individuals for their service. Veterans are invited to share their thoughts on what Veterans Day means to them. Cards, provided at all events to write personal thank you notes to Veterans, will be delivered to the Arizona State Veteran Home to show our support. Mon., Oct. 25, 10:30 a.m. to 1:30 p.m. and Tues., Oct. 26, 11:30 a.m. to 1:30 p.m. Flag Raising Ceremony Mon., Nov. 7, 8:30 to 9:30 a.m., outside A building Opening remarks from PVCC Vice President of Student Affairs Dr. Veronica Garcia, followed by the National Anthem performed by PVCC administrative assistant Aliceann Lunceford, the raising of the American flag by the USMC recruiting office Honor Guard, and the Pledge of Allegiance led by the PVCC Veterans Club. Annual Veterans Appreciation BBQ Wed., Nov. 9, 11:30 a.m. to 2 p.m., KSC Courtyard $5 donation, free for veterans and families with military identification. Proceeds benefit the Veterans Club support activities. The menu features hamburgers, hot dogs, chips, and water. Flag Raising Ceremony and Reception 7:30 a.m., Hannelly Center Members of the Marine Corps Honor Guard will perform an early morning flag raising followed by a coffee and doughnut reception inside of the college's Hannelly Center. Free and open to the public. Veteran Summit 11:00 a.m., Hacienda Room Veteran students from across the Maricopa Community Colleges District are invited to attend PC's annual Veteran Summit. Each year, the college invites local experts to provide veteran students with the tools they need to transition from the military to college to the workforce. Free and open to MCCCD students. Veteran Appreciation Fair 12:00 p.m. to 2:00 p.m., Sophomore Square The community is invited to help us say "thanks" to our veterans. The Veteran Appreciation Fair will include fun, games, and food, as well as several employers on-site who are interested in hiring veterans. Free and open to the public. Mesa Community College honors all the men and women who have served our country. On Thurs., Nov. 10, local organizations will share information on educational, recreational, and volunteer opportunities in the community 10 a.m. to 2 p.m. Local band Struck Match will perform and take requests from veterans from 11 to 11:45 a.m. Michael Somsan, a veteran and blind ironman, will tell his inspirational story and speak with attendees about his experiences and athletic accomplishments from 11:45 a.m. to 12:15 p.m. A Marine Corps birthday cake cutting will take place at 12:15 p.m. Scottsdale Community College Thurs. Nov. 10, 10:30 to 11:30 a.m., Two Waters Circle Scottsdale Community College, 9000 E. Chaparral Road, Scottsdale. Scottsdale Community College invites veterans, students and the community to a recognition ceremony for current and former military members as part of its Veterans Day celebration, Thurs., Nov. 10, 2016. Veterans will be saluted with honor, respect, and gratitude. Light refreshments will be served. For more information, contact Michelle Dew at michelle.dew(at)scottsdalecc(dot)edu. South Mountain Community College hosts a Veterans Storytelling Event featuring South Mountain's post-9/11 Veterans telling their personal stories in their own words. The Maricopa Community College District includes 10 regionally-accredited colleges–Chandler-Gilbert, Estrella Mountain, GateWay, Glendale, Mesa, Paradise Valley, Phoenix, Rio Salado, Scottsdale and South Mountain–and the Maricopa Corporate College, serving more than 100,000 students with two-year degree, certificate, and university transfer programs.
Weiler A.A.,Salt River Pima Maricopa Indian Community |
Weiler A.A.,Scottsdale Community College |
Alvar B.A.,Rocky Mountain University of Health Professions
Strength and Conditioning Journal | Year: 2013
As a result of the population explosion (termed the baby boom), there is a progressive aging of the american populace and as such additional need for allied health professionals with the knowledge, skills, and abilities to work with special populations. One of the largest categories of chronic diseases that are related to aging and lifestyle choices are cardiovascular diseases (CVDs). CVDs are the primary killer of americans. With proper training, exercise professionals have the potential to help prevent or reduce morbidity and mortality and influence financial implications for society at large. A note of importance, exercise professionals must work cooperatively with physicians and other health care professionals, and within their professional certification's scope of practice, to optimize exercise efficacy and safety. Additionally, any change in a client's symptomology warrants referral back to the physician.
Smith A.T.,Arizona State University |
Nagy J.D.,Scottsdale Community College |
Nagy J.D.,Arizona State University
Journal of Mammalogy | Year: 2015
Population resilience in a metapopulation of American pikas (Ochotona princeps) at Bodie, California, was investigated with a series of 18 detailed occupancy surveys conducted between 1989 and 2010. These were compared with earlier 1972 and 1977 censuses and earlier historical records of pikas at Bodie. There is concern that American pikas may be increasingly vulnerable to warm temperatures due to climate change, and this investigation represents the longest study of the species in a relatively low-elevation (warm) environment. The Bodie pika population represents one of the best mammalian examples of a classic metapopulation system. Annual number of observed patch extinctions (total = 114) and recolonizations (109) varied greatly among the 18 census intervals. There has been no decline in percent of patches occupied in the northern half of the study area since 1972, and the number of documented pikas in the north in recent surveys exceeded the numbers found in 1972 and 1977. In contrast, the southern half of the metapopulation collapsed during our study, apparently the result of stochasticity of metapopulation dynamics; no southern patches were occupied after 2006. The potential impact of temperature on metapopulation dynamics was examined using long-term chronic (average summer monthly maximum) and acute threshold (number of days ≥ 25°C and ≥ 28°C within a year) temperatures. There is no evidence that warming temperatures have directly and negatively affected pika persistence at Bodie. Neither warm chronic nor acute temperatures increased the frequency of extinctions of populations on patches, and relatively cooler chronic or acute temperatures did not lead to an increase in the frequency of recolonization events. Warm temperatures, however, could have impeded the dispersal of colonists moving from north to south, thus contributing to the failure of the southern region to become repopulated. © 2015 American Society of Mammalogists, .
Hews S.,Arizona State University |
Eikenberry S.,Arizona State University |
Nagy J.D.,Scottsdale Community College |
Kuang Y.,Arizona State University
Journal of Mathematical Biology | Year: 2010
Chronic hepatitis B virus (HBV) infection is a major cause of human suffering, and a number of mathematical models have examined within-host dynamics of the disease. Most previous HBV infection models have assumed that: (a) hepatocytes regenerate at a constant rate from a source outside the liver; and/or (b) the infection takes place via a mass action process. Assumption (a) contradicts experimental data showing that healthy hepatocytes proliferate at a rate that depends on current liver size relative to some equilibrium mass, while assumption (b) produces a problematic basic reproduction number. Here we replace the constant infusion of healthy hepatocytes with a logistic growth term and the mass action infection term by a standard incidence function; these modifications enrich the dynamics of a well-studied model of HBV pathogenesis. In particular, in addition to disease free and endemic steady states, the system also allows a stable periodic orbit and a steady state at the origin. Since the system is not differentiable at the origin, we use a ratio-dependent transformation to show that there is a region in parameter space where the origin is globally stable. When the basic reproduction number, R0, is less than 1, the disease free steady state is stable. When R0 > 1 the system can either converge to the chronic steady state, experience sustained oscillations, or approach the origin. We characterize parameter regions for all three situations, identify a Hopf and a homoclinic bifurcation point, and show how they depend on the basic reproduction number and the intrinsic growth rate of hepatocytes. © 2009 Springer-Verlag.
Eikenberry S.E.,Arizona State University |
Eikenberry S.E.,University of Southern California |
Nagy J.D.,Scottsdale Community College |
Kuang Y.,Arizona State University
Biology Direct | Year: 2010
Background: Androgens bind to the androgen receptor (AR) in prostate cells and are essential survival factors for healthy prostate epithelium. Most untreated prostate cancers retain some dependence upon the AR and respond, at least transiently, to androgen ablation therapy. However, the relationship between endogenous androgen levels and cancer etiology is unclear. High levels of androgens have traditionally been viewed as driving abnormal proliferation leading to cancer, but it has also been suggested that low levels of androgen could induce selective pressure for abnormal cells. We formulate a mathematical model of androgen regulated prostate growth to study the effects of abnormal androgen levels on selection for pre-malignant phenotypes in early prostate cancer development.Results: We find that cell turnover rate increases with decreasing androgen levels, which may increase the rate of mutation and malignant evolution. We model the evolution of a heterogeneous prostate cell population using a continuous state-transition model. Using this model we study selection for AR expression under different androgen levels and find that low androgen environments, caused either by low serum testosterone or by reduced 5α-reductase activity, select more strongly for elevated AR expression than do normal environments. High androgen actually slightly reduces selective pressure for AR upregulation. Moreover, our results suggest that an aberrant androgen environment may delay progression to a malignant phenotype, but result in a more dangerous cancer should one arise.Conclusions: The model represents a useful initial framework for understanding the role of androgens in prostate cancer etiology, and it suggests that low androgen levels can increase selection for phenotypes resistant to hormonal therapy that may also be more aggressive. Moreover, clinical treatment with 5α-reductase inhibitors such as finasteride may increase the incidence of therapy resistant cancers.Reviewers: This article was reviewed by Ariosto S. Silva (nominated by Marek Kimmel) and Marek Kimmel. © 2010 Eikenberry et al; licensee BioMed Central Ltd.
Nagy J.D.,Scottsdale Community College |
Armbruster D.,Arizona State University
Mathematical Biosciences and Engineering | Year: 2012
The major goal of evolutionary oncology is to explain how malignant traits evolve to become cancer "hallmarks." One such hallmark - the angiogenic switch - is difficult to explain for the same reason altruism is difficult to explain. An angiogenic clone is vulnerable to "cheater" lineages that shunt energy from angiogenesis to proliferation, allowing the cheater to outcompete cooperative phenotypes in the environment built by the cooperators. Here we show that cell- or clone-level selection is sufficient to explain the angiogenic switch, but not because of direct selection on angiogenesis factor secretion - angiogenic potential evolves only as a pleiotropic afterthought. We study a multiscale mathematical model that includes an energy management system in an evolving angiogenic tumor. The energy management model makes the counterintuitive prediction that ATP concentration in resting cells increases with increasing ATP hydrolysis, as seen in other theoretical and empirical studies. As a result, increasing ATP hydrolysis for angiogenesis can increase proliferative potential, which is the trait directly under selection. Intriguingly, this energy dynamic allows an evolutionary stable angiogenesis strategy, but this strategy is an evolutionary repeller, leading to runaway selection for extreme vascular hypo-or hyperplasia. The former case yields a tumor-on-a-tumor, or hypertumor, as predicted in other studies, and the latter case may explain vascular hyperplasia evident in certain tumor types.
Portz T.,Arizona State University |
Portz T.,University of Wisconsin - Madison |
Kuang Y.,Arizona State University |
Nagy J.D.,Scottsdale Community College
AIP Advances | Year: 2012
Prostate cancer is commonly treated by a form of hormone therapy called androgen suppression. This form of treatment, while successful at reducing the cancer cell population, adversely affects quality of life and typically leads to a recurrence of the cancer in an androgen-independent form. Intermittent androgen suppression aims to alleviate some of these adverse affects by cycling the patient on and off treatment. Clinical studies have suggested that intermittent therapy is capable of maintaining androgen dependence over multiple treatment cycles while increasing quality of life during off-treatment periods. This paper presents a mathematical model of prostate cancer to study the dynamics of androgen suppression therapy and the production of prostate-specific antigen (PSA), a clinical marker for prostate cancer. Preliminary models were based on the assumption of an androgen-independent (AI) cell population with constant net growth rate. These models gave poor accuracy when fitting clinical data during simulation. The final model presented hypothesizes an AI population with increased sensitivity to low levels of androgen. It also hypothesizes that PSA production is heavily dependent on androgen. The high level of accuracy in fitting clinical data with this model appears to confirm these hypotheses, which are also consistent with biological evidence. © 2012 Author(s).
Morken J.D.,Arizona State University |
Packer A.,Arizona State University |
Everett R.A.,Arizona State University |
Nagy J.D.,Arizona State University |
And 2 more authors.
Cancer Research | Year: 2014
For progressive prostate cancer, intermittent androgen deprivation (IAD) is one of the most common and effective treatments. Although this treatment is usually initially effective at regressing tumors, most patients eventually develop castration-resistant prostate cancer (CRPC), for which there is no effective treatment and is generally fatal. Although several biologic mechanisms leading to CRPC development and their relative frequencies have been identified, it is difficult to determine which mechanisms of resistance are developing in a given patient. Personalized therapy that identifies and targets specific mechanisms of resistance developing in individual patients is likely one of the most promising methods of future cancer therapy. Prostate-specific antigen (PSA) is a biomarker for monitoring tumor progression. We incorporated a cell death rate (CDR) function into a previous dynamical PSA model that was highly accurate at fitting clinical PSA data for 7 patients. The mechanism of action of IAD is largely induction of apoptosis, and each mechanism of resistance varies in its CDR dynamics. Thus, we analyze the CDR levels and their time-dependent oscillations to identify mechanisms of resistance to IAD developing in individual patients. ©2014 AACR.