Plaza Medical Center

Fort Worth, TX, United States

Plaza Medical Center

Fort Worth, TX, United States
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PubMed | Plaza Medical Center and Fort Worth Transplant Institute
Type: | Journal: Case reports in nephrology | Year: 2015

Renal transplant vein stenosis is a rare cause of allograft dysfunction. Percutaneous stenting appears to be safe and effective treatment for this condition. A 56-year-old Caucasian female with end stage renal disease received a deceased donor renal transplant. After transplant, her serum creatinine improved to a nadir of 1.2mg/dL. During the third posttransplant month, her serum creatinine increased to 2.2mg/dL. Renal transplant biopsy showed BK nephropathy. Mycophenolate was discontinued. Over the next 2 months, her serum creatinine crept up to 6.2mg/dL. BK viremia improved from 36464 copies/mL to 15398 copies/mL. A renal transplant ultrasound showed lower pole arteriovenous fistula and abnormal waveforms in the renal vein. Carbon dioxide (CO2) angiography demonstrated severe stenosis of the transplant renal vein. Successful coil occlusion of fistula was performed along with angioplasty and deployment of stent in the renal transplant vein. Serum creatinine improved to 1.5mg/dL after.


The International Association of HealthCare Professionals is pleased to welcome Que Thu Pravong, DO, Family Medicine Physician, to their prestigious organization with her upcoming publication in The Leading Physicians of the World. Dr. Que Thu Pravong is a highly trained and qualified family practitioner with an extensive expertise in all facets of her work. Dr. Pravong has been in practice for more than 13 years and is currently serving patients within Lifetime Family Medicine in Flower Mound, Texas. Furthermore, she is also affiliated with Texas Health Presbyterian Hospital in Dallas. Dr. Que Thu Pravong graduated with her Doctorate of Osteopathic Medicine from the University of North Texas Health Science Center. Following her graduation, she completed her residency at Plaza Medical Center of Fort Worth. Board certified in Family Medicine, Dr. Pravong keeps up to date with the latest advances in her field by maintaining a professional membership with the American Osteopathic Association, the Texas Medical Association, and the Texas Osteopathic Medical Association. She attributes her success to her commitment to listening to her patients, and in her free time, Dr. Pravong enjoys reading, working out, and spending time with her family and friends. Learn more about Dr. Pravong here: https://www.thpg.org/lifetimefamilymedicine/pages/default.aspx and be sure to read her upcoming publication in The Leading Physicians of the World. FindaTopDoc.com is a hub for all things medicine, featuring detailed descriptions of medical professionals across all areas of expertise, and information on thousands of healthcare topics.  Each month, millions of patients use FindaTopDoc to find a doctor nearby and instantly book an appointment online or create a review.  FindaTopDoc.com features each doctor’s full professional biography highlighting their achievements, experience, patient reviews and areas of expertise.  A leading provider of valuable health information that helps empower patient and doctor alike, FindaTopDoc enables readers to live a happier and healthier life.  For more information about FindaTopDoc, visit http://www.findatopdoc.com


News Article | December 16, 2016
Site: www.prweb.com

Healthcare Associates of Texas announced today the addition of Jason Conn, DO, to its primary care practice in Coppell, TX. Dr. Conn has been practicing medicine in the Dallas-Fort Worth area since 2003 and will be working alongside Charles Calvert, DO. “Dr. Conn is a top-notch physician who will be an outstanding addition to our team of well-trained, well-spoken and caring physicians,” said Dr. Charles Powell, chief of clinical operations. “His advanced skill set, combined with our technology, was a perfect match.” Dr. Conn earned a bachelor of arts in biology from Baylor University and a doctor of osteopathic medicine from the University of North Texas Health Science Center. Dr. Conn completed his internship and internal medicine residency at Plaza Medical Center in Fort Worth. “I am excited to join Healthcare Associates of Texas and to have the resources of a well-established, successful, respected and cutting-edge practice behind me,” said Dr. Conn. “It is also an honor to be able to practice quality medicine in a tightly knit community.” Dr. Conn is board certified in Internal Medicine by the American Osteopathic Board of Internal Medicine and is a member of the American Osteopathic Association. He has been a member of the American College of Osteopathic Internists, Texas Osteopathic Medical Association, Texas Medical Association, American Medical Association, and Christian Medical and Dental Society. About Healthcare Associates of Texas Healthcare Associates of Texas LLC was founded in 1991 and is headquartered in Irving, Texas. The company currently has three north Texas locations. In addition to offering primary care and family medicine services, the company provides patients with a number of ancillary services, including imaging, diagnostics, physical medicine and an in-house pharmacy.


Rajendiran S.,University of North Texas Health Science Center | Parwani A.V.,UPMC Shadyside Hospital | Hare R.J.,Plaza Medical Center | Dasgupta S.,University of North Texas Health Science Center | And 2 more authors.
Molecular Cancer | Year: 2015

Background: MicroRNAs (miRNAs) are crucial molecules that regulate gene expression and hence pathways that are key to prostate cancer progression. These non-coding RNAs are highly deregulated in prostate cancer thus facilitating progression of the disease. Among the many genes that have gained importance in this disease, Migration and invasion enhancer 1 (MIEN1), a novel gene located next to HER2/neu in the 17q12 amplicon of the human chromosome, has been shown to enhance prostate cancer cell migration and invasion, two key processes in cancer progression. MIEN1 is differentially expressed between normal and cancer cells and tissues. Understanding the regulation of MIEN1 by microRNA may enable development of better targeting strategies. Methods: The miRNAs that could target MIEN1 were predicted by in silico algorithms and microarray analysis. The validation for miRNA expression was performed by qPCR and northern blotting in cells and by in situ hybridization in tissues. MIEN1 and levels of other molecules upon miRNA regulation was determined by Western blotting, qPCR, and immunofluorescence. The functional effects of miRNA on cells were determined by wound healing cell migration, Boyden chamber cell invasion, clonal and colony formation assays. For knockdown or overexpression of the miRNA or overexpression of MIEN1 3'UTR, cells were transfected with the oligomiRs and plasmids, respectively. Results: A novel miRNA, hsa-miR-940 (miR-940), identified and validated to be highly expressed in immortalized normal cells compared to cancer cells, is a regulator of MIEN1. Analysis of human prostate tumors and their matched normal tissues confirmed that miR-940 is highly expressed in the normal tissues compared to its low to negligible expression in the tumors. While MIEN1 is a direct target of miR-940, miR-940 alters MIEN1 RNA, in a quantity as well as cell dependent context, along with altering its downstream effectors. The miR-940 inhibited migratory and invasive potential of cells, attenuated their anchorage-independent growth ability, and increased E-cadherin expression, implicating its role in mesenchymal-to-epithelial transition (MET). Conclusions: These results, for the first time, implicate miR-940, a regulator of MIEN1, as a promising novel diagnostic and prognostic tool for prostate cancer. © 2014 Rajendiran et al.; licensee BioMed Central Ltd.


Rajendiran S.,University of North Texas Health Science Center | Parwani A.V.,UPMC Shadyside Hospital | Hare R.J.,Plaza Medical Center | Dasgupta S.,University of North Texas Health Science Center | And 4 more authors.
Molecular Cancer | Year: 2014

Background: MicroRNAs (miRNAs) are crucial molecules that regulate gene expression and hence pathways that are key to prostate cancer progression. These non-coding RNAs are highly deregulated in prostate cancer thus facilitating progression of the disease. Among the many genes that have gained importance in this disease, Migration and invasion enhancer 1 (MIEN1), a novel gene located next to HER2/neu in the 17q12 amplicon of the human chromosome, has been shown to enhance prostate cancer cell migration and invasion, two key processes in cancer progression. MIEN1 is differentially expressed between normal and cancer cells and tissues. Understanding the regulation of MIEN1 by microRNA may enable development of better targeting strategies. Methods: The miRNAs that could target MIEN1 were predicted by in silico algorithms and microarray analysis. The validation for miRNA expression was performed by qPCR and northern blotting in cells and by in situ hybridization in tissues. MIEN1 and levels of other molecules upon miRNA regulation was determined by Western blotting, qPCR, and immunofluorescence. The functional effects of miRNA on cells were determined by wound healing cell migration, Boyden chamber cell invasion, clonal and colony formation assays. For knockdown or overexpression of the miRNA or overexpression of MIEN1 3'UTR, cells were transfected with the oligomiRs and plasmids, respectively. Results: A novel miRNA, hsa-miR-940 (miR-940), identified and validated to be highly expressed in immortalized normal cells compared to cancer cells, is a regulator of MIEN1. Analysis of human prostate tumors and their matched normal tissues confirmed that miR-940 is highly expressed in the normal tissues compared to its low to negligible expression in the tumors. While MIEN1 is a direct target of miR-1, miR-940 alters MIEN1 RNA, in a quantity as well as cell dependent context, along with altering its downstream effectors. The miR-940 inhibited migratory and invasive potential of cells, attenuated their anchorage-independent growth ability, and increased E-cadherin expression, implicating its role in mesenchymal-to-epithelial transition (MET). Conclusions: These results, for the first time, implicate miR-940, a regulator of MIEN1, as a promising novel diagnostic and prognostic tool for prostate cancer. © Rajendiran et al.; licensee BioMed Central Ltd.


Rajendiran S.,University of North Texas Health Science Center | Kpetemey M.,University of North Texas Health Science Center | Maji S.,University of North Texas Health Science Center | Gibbs L.D.,University of North Texas Health Science Center | And 5 more authors.
Cancer Biology and Therapy | Year: 2015

Oral squamous cell carcinoma is a highly malignant tumor with the potential to invade local and distant sites and promote lymph node metastasis. Major players underlying the molecular mechanisms behind tumor progression are yet to be fully explored. Migration and invasion enhancer 1 (MIEN1), a novel protein overexpressed in various cancers, facilitates cell migration and invasion. In the present study we investigated the expression and role of MIEN1 in oral cancer progression using an in vitro model, patient derived oral tissues and existing TCGA data. Expression analysis using immortalized normal and cancer cells demonstrated increased expression of MIEN1 in cancer. Assays performed after MIEN1 knockdown in OSC-2 cells showed decreased migration, invasion and filopodia formation; while MIEN1 overexpression in DOK cells increased these characteristics and also up-regulated some Akt/NF-κB effectors, thereby suggesting an important role for MIEN1 in oral cancer progression. Immunohistochemical staining and analyses of oral tissue specimens, collected from patients over multiple visits, revealed significantly more staining in severe dysplasia and squamous cell carcinoma compared to mildly dysplastic or hyperplastic tissues. Finally, this was corroborated with the TCGA dataset, where MIEN1 expression was not only higher in intermediate and high grade cancer with significantly lower survival but also correlated with smoking. In summary, we demonstrate that MIEN1 expression not only positively correlates with oral cancer progression but also seems to be a critical molecular determinant in migration and invasion of oral cancer cells, thereby, playing a possible role in their metastatic dissemination. © 2015 Taylor & Francis Group, LLC.


PubMed | Plaza Medical Center
Type: Journal Article | Journal: Gastroenterology research | Year: 2016

The standard endoscopic ultrasound (EUS) approach of cystogastrostomy involves the use of series of plastic pigtail stents that are placed through the wall of the cyst. The use of a single stent has also been described in the literature. Here we describe five cases of EUS-guided cystogastrostomy with irrigation of infected pancreatic pseudocysts using a single self-expandable metal stent (SEMS). To our knowledge this has not been described in the literature in the United States. This novice approach will have significant implications in the management of infected pseudocysts with a lower morbidity, mortality and overall cost compared to conventional management such as surgery or percutaneous drainage.


PubMed | Plaza Medical Center
Type: Journal Article | Journal: Journal of nursing care quality | Year: 2012

This article presents the implementation and 1-year evaluation of a staff nurse-driven initiative addressing a Joint Commission patient safety imperative to improve the consistency of handoff communication. An interdepartmental ticket was developed and implemented through a planned process. The interdepartmental ticket serves as a requisite passport for providing key communication of critical information to facilitate a safe transition between medical-surgical and progressive care nursing units and specific ancillary departments.


Yurvati A.H.O.,University of North Texas Health Science Center | Thompson J.B.,University of North Texas Health Science Center | Woods T.N.,University of North Texas Health Science Center | Woods T.N.,Plaza Medical Center
Journal of the American Osteopathic Association | Year: 2013

Splenosis is a rare occurrence that is defined as autotransplantation of splenic tissue usually after splenic rupture due to trauma and subsequent splenectomy. Although splenosis most commonly occurs in the abdomen, the authors report a rare case of thoracic splenosis after remote thoracoabdominal trauma. A 62-year-old woman was found to have lower-lobe, pleural-based nodular lesions in juxtaposition to the posteromedial segment of the lung during workup for an abdominal hernia. Surgical excision of the mass confirmed the diagnosis of ectopic splenic tissue, and splenosis was diagnosed. This woman was among the rare 18% of people who are found to have splenosis in the intrathoracic space. In the workup of pulmonary nodules in patients with a history of trauma, splenosis should be a consideration. © 2013 American Osteopathic Association.


PubMed | The Texas Institute, Plaza Medical Center and A senior consultant at
Type: Journal Article | Journal: Nursing management (Harrow, London, England : 1994) | Year: 2016

ALL AROUND the world, countries currently face the most severe nursing shortage in history ( Hatcher 2006 ). This shortage stems partly from the effect on employees, and ultimately therefore on organisations ( Judkins and Furlow 2004 ), of work related stress.

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