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Lubicz Górny, Poland

Jeczmionek L.,Oil and Gas Institute
Polish Journal of Environmental Studies | Year: 2014

This publication presents the results of research concerning an improvement of low-temperature properties of HVO paraffinic biocomponents by utilizing zeoformed waste natural oils. The process was performed using a special zeolite catalyst based on the hydrogen form of the ZSM-5 zeolites. This method is generally recognized as a new approach for obtaining low-temperature performance improvements of the HVO biocomponents.


Steliga T.,Oil and Gas Institute
Polish Journal of Environmental Studies | Year: 2012

Petroleum substances are the main source of pollutants stored in old waste pits. They cause degradation of biological life in the area of storage. The aim of this article is to present laboratory research on petroleum pollutant biodegradation. The hydrocarbon contaminants in old drilling wastes came from the Graby-19 waste pit. The tests included basic bioremediation and inoculation with a biopreparation based on non-pathogenic species of bacteria and fungi, which were identified in molecular research. Multicriteria effectiveness estimation of petroleum pollutant biodegradation in the tested wastes enabled determination of a role of fungi in this process. Biopreparation enrichment was done in the final phase with non-pathogenic fungi isolated from the waste during purification. As a result, an increase in long-chain n-alkanes and aromatic hydrocarbon (BTEX and PAH) biodegradation was observed. Biodegradation rate constants for petroleum hydrocarbons, calculated on the basis of a mathematical model, can serve as proof of fungi usage in the bioremediation process.


Maes M.,Maes Clinics at TRIA | Kubera M.,Polish Academy of Sciences | Obuchowiczwa E.,University of Silesia | Goehler L.,University of Virginia | Brzeszcz J.,Oil and Gas Institute
Neuroendocrinology Letters | Year: 2011

There is now evidence that depression, as characterized by melancholic symptoms, anxiety, and fatigue and somatic (F&S) symptoms, is the clinical expression of peripheral cell-mediated activation, inflammation and induction of oxidative and nitrosative stress (IO&NS) pathways and of central microglial activation, decreased neurogenesis and increased apoptosis. This review gives an explanation for the multiple "co-morbidities" between depression and a large variety of a) brain disorders related to neurodegeneration, e.g. Alzheimer's, Parkinson's and Huntington's disease, multiple sclerosis and stroke; b) medical disorders, such as cardiovascular disorder, chronic fatigue syndrome, chronic obstructive pulmonary disease, rheumatoid arthritis, psoriasis, systemic lupus erythematosus, inflammatory bowel disease, irritable bowel syndrome, leaky gut, diabetes type 1 and 2, obesity and the metabolic syndrome, and HIV infection; and c) conditions, such as hemodialysis, interferon-a-based immunotherapy, the postnatal period and psychosocial stressors. The common denominator of all those disorders/ conditions is the presence of microglial activation and/or activation of peripheral IO&NS pathways. There is evidence that shared peripheral and / or central IO&NS pathways underpin the pathophysiology of depression and the previously mentioned disorders and that activation of these IO&NS pathways contributes to shared risk. The IO&NS pathways function as a smoke sensor that detect threats in the peripheral and central parts of the body and signal these threats as melancholic, anxiety, and fatigue and somatic (F&S) symptoms. The presence of concomitant depression is strongly associated with a lower quality of life and increased morbidity and mortality in medical disorders. This may be explained since depression contributes to increased (neuro)inflammatory burden and may therefore drive the inflammatory and degenerative progression. It is concluded that the activation of peripheral and / or central IO&NS pathways may explain the co-occurrence of depression with the above disorders. This shows that depression belongs to the spectrum of inflammatory and degenerative disorders. ©2011 Neuroendocrinology Letters.


Jeczmionek L.,Oil and Gas Institute | Porzycka-Semczuk K.,Cracow University of Technology
Fuel | Year: 2013

Initial results for the zeoforming of triglycerides from rapeseed oil and their application for the production of second generation biocomponents were described. It was found that the cloud points of the hydroraffinates obtained from a kerosene (boiling range 160-245 C) and zeoformed rapeseed oil feedstock were lower (by several degrees Centigrade) than those of the hydroraffinates obtained from the unmodified rapeseed oil. This phenomenon was caused by the modification of fatty acid triglyceride structures by isomerisation of the fatty acid chains. The obtained results indicate that zeoforming may be an interesting solution for improving the low temperature properties of second generation biocomponents obtained from natural oils and/or fats by hydroconversion and co-processing. A complete understanding of the zeoforming mechanism for fatty acid triglycerides requires further research. © 2013 Elsevier Ltd. All rights reserved.


Steliga T.,Gas Technology Institute | Jakubowicz P.,Gas Technology Institute | Kapusta P.,Oil and Gas Institute
Bioresource Technology | Year: 2012

Bioremediation of weathered drill wastes severely contaminated with total petroleum hydrocarbons (TPH) (90,000-170,000mgkg-1) and BTEX (51.2-95.5mgkg-1) to soil standards was achieved over a 3-year period in three phases: initial remediation, basic bioremediation and inoculation with a biopreparation. Fourteen non-pathogenic indigenous bacteria species belonging mainly to the Actinomycetales were identified and shown to be able to degrade 63-75% of nC9-nC20, 36-51% of nC21-nC36, 36% of BTEX and 20% of PAHs (polycyclic aromatic hydrocarbons). Addition of five non-pathogenic fungi species to the bacterial consortium allowed degradation of 69-89% of nC9-nC20, 47-80% of nC21-nC36, 76% of BTEX, and 68% of PAHs. Microtox, Ostacodtoxkit, Phytotoxkit and Ames tests indicated that changes in toxicity were not connected with the decrease in TPH contents, possibly due to the formation of toxic indirect metabolites during bioremediation. No toxicity was found in the soil after bioremediation. © 2012 Elsevier Ltd.

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