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Old Westbury, NY, United States

New York Institute of Technology is a global private, independent, nonprofit, non-sectarian, coeducational research university. NYIT has five schools and two colleges, all with a strong emphasis on technology and applied scientific research: School of Architecture and Design, School of Education, School of Engineering and Computing science, School of Health Professions, School of Management, College of Arts and science and College of Osteopathic Medicine. The university has two New York campuses, one in Old Westbury Long Island and one near Columbus Circle in Manhattan, as well as several global campuses in: Abu Dhabi, United Arab Emirates; Nanjing, China; and Vancouver, Canada. New York Institute of Technology offers over 90 degree programs, including undergraduate, graduate, and professional degrees, in more than 50 fields of study, including architecture and design; arts and science; education; engineering and computing science; health professions; management; and osteopathic medicine. Its Carnegie Classification is Masters-Granting "Research University," very high research activity.NYIT students represent nearly all 50 U.S. states and 109 countries. NYIT consistently ranks in the "top 50" among U.S. universities in the north, as compiled by U.S. News & World Report. Wikipedia.


Awartani B.,New York Institute of Technology | Maghyereh A.I.,United Arab Emirates University
Energy Economics | Year: 2013

This article exploits a new spillover directional measure proposed by Diebold and Yilmaz (2009, 2012) to investigate the dynamic spillover of return and volatility between oil and equities in the Gulf Cooperation Council Countries during the period 2004 to 2012. Our results indicate that return and volatility transmissions are bi-directional, albeit asymmetric. In particular, the oil market gives other markets more than it receives in terms of both returns and volatilities. These trends were more pronounced in the aftermath of the Global Financial Crisis in 2008 as the net contribution of oil has intensified after a burst during the crisis. The empirical evidence from the sample is consistent with a system in which oil is playing the dominant role in the information transmission mechanism between oil and equities in the GCC countries. © 2012 Elsevier B.V. Source


Li K.,New York Institute of Technology
IEEE Transactions on Control Systems Technology | Year: 2013

A new robust proportional-integrative-derivative (PID) tuning method based on nonlinear optimization is developed. The closed-loop bandwidth is maximized for specified gain and phase margins (GPM) with constraint on overshoot ratio, so that criteria of closed-loop performance and robustness are both satisfied. The equations of open-loop amplitude ratio and phase change are derived based on frequency analysis for the first-order plus time delay system with a PID controller in parallel form. The equation of closed-loop amplitude ratio is also explicitly given. The formulated optimization problem from the tuning method based on GPM with constraint on closed-loop amplitude ratio is further given. The method is demonstrated in simulation examples and compared with previous work on this topic. © 1993-2012 IEEE. Source


Kobayashi S.,New York Institute of Technology
Biological and Pharmaceutical Bulletin | Year: 2015

Autophagy is a degradation system for intracellular components. One of the roles of autophagy is the prompt removal of damaged organelles. Another unique role is to supply resources that maintain metabolism in response to the cellular nutritional state. Precise management of all the components in the autophagic system is essential for cellular health. Especially important are the selectivity of target cargos for autophagy, and the coordination of autophagy with the lysosomal catabolic process. This review outlines our current understanding of autophagy and discusses potential therapeutic perspectives. Emphasis will be given to lyso-somal function as a central controller of metabolism, and to selective autophagy as a key mechanism for the efficient removal of dysfunctional organelles. © 2015 The Pharmaceutical Society of Japan. Source


Martin Gerdes A.,New York Institute of Technology
American Journal of Physiology - Heart and Circulatory Physiology | Year: 2015

The link between low thyroid hormone (TH) function and heart failure is reviewed in the present report. The idea that TH dysfunction may contribute to diseases leading to HF has been discussed for over 60 yr. A growing body of evidence from animal and human studies, particularly in recent years, suggests that TH treatment may improve clinical outcomes. Indeed, if a similar amount of positive information were available for a newly developed heart drug, there is little doubt that large-scale clinical trials would be underway with considerable excitement. THs offer the promise of improving ventricular contraction and relaxation, improving coronary blood flow, and inhibiting atherosclerosis, and new results suggest they may even reduce the incidence of arrhythmias in heart diseases. Are the potential clinical benefits worth the risk of possible overdosing? After so many years, why has this question not been answered? Clearly, the concept has not been disproven. This review explores the body of clinical evidence related to TH dysfunction and heart failure, discuss insights into pathophysiological, cellular, and molecular mechanisms provided by animal research, and discuss what is needed to resolve this long-standing issue in cardiology and move forward. © 2015 the American Physiological Society. Source


Gerdes A.M.,New York Institute of Technology
American Journal of Physiology - Heart and Circulatory Physiology | Year: 2015

By the mid-1990s, experts realized that drugs leading to improved ventricular remodeling were doing something remarkable in cardiac patients. The “age of cardiac remodeling” had begun. This created an experimental need for high-quality assessment of changes in cardiac tissue composition, including myocyte shape, myocardial fibrosis/collagen, and vascular remodeling. Many working in the field today have little or no training related to recognition of fixation artifacts or common errors associated with quantitative morphology. Unfortunately, such skills had become somewhat of a lost art during the ages of cardiac physiology in the mid-20th century and molecular biology, gaining prominence by the mid-1970s. Consequently, cardiac remodeling studies today are often seriously flawed to the point where data are not reproducible and subsequent researchers may be chasing the molecular basis of a nonexistent or erroneous phenotype. The current unacceptably high incidence of irreproducible data is a serious waste of time and resources as recently noted in comments by the National Institutes of Health director. The goal of this “how to” article is to share some lessons I have learned during nearly 40 years of assessing morphological changes in the heart. It is possible for any laboratory to routinely publish highly reproducible morphological data that stand the test of time and contribute to our fundamental knowledge of cardiac remodeling and the molecular mechanisms that drive it. © 2015 the American Physiological Society. Source

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