Villa General Mitre, Argentina

John F. Kennedy Argentine University
Villa General Mitre, Argentina

The Universidad Argentina John F. Kennedy was founded by Miguel Herrera Figueroa, JD, on April 4, 1964 in Buenos Aires, Argentina. The university has 18 units in the City of Buenos Aires and two in Buenos Aires Province. In 2008, it had 39 academic departments and a students department, where activities related to sports are developed with the Kennedy Choir and the Theater Workshop. Wikipedia.

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PubMed | CONICET, University of Greifswald, University of Malaga, Pontifical Xavierian University and John F. Kennedy Argentine University
Type: | Journal: Neuroscience letters | Year: 2014

Perinatal asphyxia represents an important cause of severe neurological deficits including delayed mental and motor development, epilepsy, major cognitive deficits and blindness. However, at the moment, most of the therapeutic strategies were not well targeted toward the processes that induced the brain injury during perinatal asphyxia. Traditionally, experimental research focused on neurons, whereas astrocytes have been more related with the damage mechanisms of perinatal asphyxia. In this work, we propose to review possible protective as well as deleterious roles of astrocytes in the asphyctic brain with the aim to stimulate further research in this area of perinatal asphyxia still not well studied.

Cosentino J.C.,John F. Kennedy Argentine University
Agora (Brazil) | Year: 2013

This is a critical and comparative reading of the manuscripts of The Ego and the Id. We can read in this work a dissymmetry between the repressed unconscious and the material that remains unknown. We depart from chapter IV, "The two kinds of Drives". As Freud announces, the matters he displays resume thoughts from his writings of 1920. So, starting with the redefinition of Ucs and afterwards, with the reformulation of the idea of Todestrieb, they remain much closer to psychoanalysis than Beyond the pleasure principle. But what he calls, "our speculation" returns in the conclusions of chapter IV about the assumption of the Todestrieb. With the breaking through of the not bound: what are the purposes the Todestrieb serves, when, as he says in 1923, it has its hands free? In 1924 he confirms that not-all the drive is inscribed in the representation. The silence of the drive intervenes: this border, where id speaks, id enjoys and knows nothing.

Catanzaro O.,John F. Kennedy Argentine University | Capponi J.A.,John F. Kennedy Argentine University | Michieli J.,John F. Kennedy Argentine University | Labal E.,John F. Kennedy Argentine University | And 2 more authors.
Peptides | Year: 2013

Diabetic peripheral neuropathy is one the most common complications of diabetes mellitus and frequently results in clinically significant morbidities such as pain, foot ulcers and amputations. The diabetic condition progresses from early functional changes to late, poorly reversible structural changes. The chronic hyperglycemia measured alongside diabetes development is associated with significant damage and failure of various organs. In the present study diabetes was induced in male Wistar rats by a single dose of streptozotocin (STZ) and the association between the BKB1-R and the oxidative stress and Na+-K+ ATPase activity in nervous tissues was analysed. The results showed that the resulting hyperglycemia induced a reduction of the neuronal electrical function integrity and increased oxidative stress in the sciatic nerve homogenates of 30 days diabetic rats. Malondialdehyde (MDA) used as a marker of oxidative stress was elevated whereas Biological Antioxidant Potential (BAP), glutathion (GSH) levels and superoxide dismutase (SOD) activity were decreased. Treatment of the rats 3 days before the end of the 4 week period with the BKB1 antagonist R-954 restored the neuronal activity and significantly attenuated the oxidative stress as shown by the level of the various markers returning close to levels found in control rats. Our results suggest that the BKB1-R subtype is overexpressed in sciatic nerve during the STZ-induced diabetes development as evidenced by inhibitory effects of the BKB1-R antagonist R-954. The beneficial role of BKB 1-R antagonist R-954 for the treatment of diabetic neuropathy is also suggested. © 2013 Elsevier Inc. All rights reserved.

Catanzaro O.L.,John F. Kennedy Argentine University | Catanzaro O.L.,University of Salvador | Dziubecki D.,John F. Kennedy Argentine University | Obregon P.,John F. Kennedy Argentine University | And 2 more authors.
Neuropeptides | Year: 2010

Insulin-dependent diabetes mellitus (type 1 diabetes) is an inflammatory autoimmune disease associated with many complications including nephropathy, retinopathy, neuropathy and hyperalgesia. Experimental evidence has shown that the bradykinin B1 receptor (BKB1-R) is involved in the development of type 1 diabetes and found to be upregulated alongside the disease. In the present study the effects of the selective BKB1-R antagonist the R-954 (Ac-Orn-[Oic2, α-MePhe5, D-β Nal7, Ile8 ]des-Arg9-BK and the BKB1-R agonist des Arg9-BK (DBK) were studied on diabetic hyperglycemia. Diabetic type 1 was induced in C57 BL/KsJ mdb male mice by five consecutives doses of STZ (45 mg/kg i.p.). A glucose tolerance test (GTT) was performed by an intraperitoneal administration of glucose, 8, 12 and 18 days after the diabetes induction. The induction of type 1 diabetes provoked a significant hyperglycemia levels in diabetic mice at 12 and 18 days after STZ. The administration of R-954 (400 μg/kg i.p.) at 12 and 18 days after STZ returned the glycemia levels of this animals to normal values. In addition the administration of DKB (300 μg/kg i.p.) significantly potentiated the diabetes-induced hyperglycemia; this effect that was totally reversed by R-954. These results provide further evidence for the implication of BKB1-R in the type 1 diabetes mellitus (insulitis). © 2009.

Costa N.E.,John F. Kennedy Argentine University
Vertex (Buenos Aires, Argentina) | Year: 2012

This article is intended to try give light to one of the questions that often reveals to the student of Psychology and Psychiatry on the application of Jungian psychotherapy. How is its praxis? Is it done face to face or does it use a couch? Which are the methods employed? On the basis of Jung's own claims that it defends no doctrine and only describes facts and leaves to everyone the freedom to deal with them, we will find from a fundamentally empirical analytic space. It does not work from a scheme systematized on the basis of a prior theoretical orthodoxy. On the contrary, each patient will be presented as a new challenge to the understanding of the therapist.

Costa N.E.,John F. Kennedy Argentine University
Vertex (Buenos Aires, Argentina) | Year: 2012

Jung conceptualized the collective unconscious as the locus of the myths, the absolutely genuine and primary patterns of thinking and feeling of humanity. The legend of Oedipus or myth of incest is a irrefutable proof of its eternal validity. The present essay is an attempt to take an alternative look to a history that belong to all of us. Situations that are developed in this drama, like the characters involved in it may charge significance as an archetypal and essentially symbolic interpretation.

Avella B.,John F. Kennedy Argentine University | Avella B.,Systemes Humains Machines Inc. Latin America Division
Canadian Nuclear Society - 32nd Annual Conference of the Canadian Nuclear Society and 35th CNS/Can Student Conference 2011 | Year: 2011

This paper reviews the notion of safety culture and then describes some of the tools that can be used to assess it. Required characteristics to obtain reliable tools and techniques are provided, along with a short summary of the most common and important tools and techniques used to assess safety culture at the nuclear field is described. At the end of this paper, the reader will better understand the importance of the safety culture of the organization and will have requirements to help him in choosing reliable tools and techniques. Further, there will be recommendations on how best to follow-up after an assessment of safety culture.

Kolliker Frers R.A.,University of Buenos Aires | Kolliker Frers R.A.,J M Ramos Mejia Hospital | Bisoendial R.J.,University of Amsterdam | Montoya S.F.,J M Ramos Mejia Hospital | And 6 more authors.
IJC Metabolic and Endocrine | Year: 2015

Introduction and background: In the last few years, a substantial body of evidence indicates that cutaneous psoriasis and psoriatic arthritis patients are at higher risk of developing cardiovascular disease. However, underlying mechanism remains not completely understood. In this review we discuss the role of the immune system in the development of atherosclerosis, focusing on available data implicating the role of an enhanced immune-mediated proinflammatory status in psoriasis and psoriatic arthritis diseases. Methods: A systematic search was performed on Pubmed until November 2014, with preference to the sources published within the past 8. years, including epidemiological studies (prospective and retrospective); cross-sectional case-control studies and reviews. Articles were selected according critical associations using the following keywords: arthritis, immune-mediated inflammatory diseases, and psoriasis. These were combined with closely related keywords reflecting cardiovascular diseases: atherogenesis, endothelial dysfunction, intima media thickness, subclinical atherosclerosis, plaque, thrombosis, thrombus, fibrinolysis, coagulation, and reactive oxygen species. Both types of disease selected terms were separately combined with non-traditional (innate and adaptive pro and anti-inflammatory immune molecules and cells) and traditional (metabolic related conditions and molecules) cardiovascular risk factors. Results and conclusions: Psoriasis and psoriatic arthritis diseases illustrate that immune-mediated activated crossroads of inflammation beyond enhanced cardiovascular risk factors are the result of an interplay between different proatherogenic mediators derived from metabolic, vascular and autoimmune joint and skin inflamed target tissue. Consistent with this point of view, psoriasis and psoriatic arthritis diseases offer an invaluable opportunity to reinforce our knowledge about atherosclerotic cardiovascular disease. © 2015.

Catanzaro O.,John F. Kennedy Argentine University | Catanzaro O.,University of Salvador | Labal E.,John F. Kennedy Argentine University | Andornino A.,John F. Kennedy Argentine University | And 3 more authors.
Peptides | Year: 2012

The chronic hyperglycemia measured alongside diabetes development is associated with significant long-term damage and failure of various organs. In the present study it was shown that hyperglycemia induced early and long term increases in nitric oxide (NO) levels, kallikrein activity and vascular capillary permeability measured as plasma extravasation, and decreases of Na/K ATPase activity in diabetic rat retina 4 and 12 weeks after streptozotocin (STZ) injection. Treatment of the animals for 5 consecutive days with a novel selective bradykinin B 1 receptor (BKB 1-R) antagonist R-954 (2 mg/kg s.c) at the end of the 4 and 12 week periods highly reduced NO, kallikrein and capillary permeability and increased Na/K ATPase activity in the retina. These results suggest that the BKB 1-R receptor subtype is over-expressed during the streptozotocin-induced development of diabetes in rat retina as evidenced by the inhibitory effects of the BKB 1-R antagonist R-954 on NO, kallikrein and vascular permeability increases as well as Na/K ATPase decreases. The beneficial role of the BKB 1-R antagonist R-954 for the treatment of the diabetic retinopathy is also suggested. © 2012 Elsevier Inc. All rights reserved.

PubMed | Fundacion Cientifica Buenos Aires, John F. Kennedy Argentine University and Columbia University
Type: Journal Article | Journal: Clinical implant dentistry and related research | Year: 2016

To measure the buccal plate reconstruction of extraction sockets with labial plate dehiscence defects using a bone allograft in combination with an absorbable collagen membrane and a custom-healing abutment at the time of tooth removal.Ten patients underwent immediate implant placement and reconstruction of the buccal plate. Cone beam computed tomography (CBCT) was performed preextraction, immediately after bone grafting and implant placement (day 0), and between 6 and 9 months following implant surgery. Measurements were taken at three levels: coronal (L1), middle (L2), and apical (L3) level.Implants placed into sockets with labial plate dehiscence defects demonstrated radiographic reformation of the labial plate dehiscence defect at 6 to 9 months posttreatment. The net gain in labial plate on cone beam computerized tomography (CBCT) in L1 and L2 was 3.0mm, where 0mm existed at pretreatment. The minimum amount of labial plate thickness of 2.0mm was achieved in all treated sites, evaluated radiographically at 6 to 9 months postoperatively, in a single procedure, without flap elevation and maintaining the gingival architecture and satisfactory esthetics.Placing an absorbable membrane, bone graft, and custom-healing abutment at the time of flapless anterior tooth extraction and immediate implant placement into a socket with a labial osseous dehiscence is a viable clinical technique to reconstitute the absence of the labial bone plate.

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