Bila Tserkva National Agrarian University

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Bila Tserkva, Ukraine
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Agency: European Commission | Branch: FP7 | Program: CP-FP | Phase: KBBE-2008-1-2-01 | Award Amount: 3.92M | Year: 2009

Organic and low-input farming systems have been shown to benefit farmland biodiversity although a generic indicator system to assess these benefits at the European level is lacking. The BIOBIO project will therefore pursue the following objectives: 1. Conceptualization of criteria for a scientifically-based selection of biodiversity indicators for organic/low-input farming systems; 2. Assessment and validation of a set of candidate biodiversity indicators in representative case studies across Europe (and in ICPC countries); 3. Preparation of guidelines for the implementation of biodiversity indicators for organic/low-input farming systems for Europe and beyond. Existing indirect farm management indicators as well as direct indicators for genetic, species and habitat diversity will be assessed for their scientific soundness, practicality, geographic scope and usefulness for stakeholders. Candidate indicators will be tested in a standardised design in twelve case studies across Europe and later in three ICPC countries. Case study regions will include pannonian, alpine, boreal, Atlantic and Mediterranean grassland systems (both organic and/or low-input), rain fed organic farms under temperate and Mediterranean conditions, mixed organic farming, organic special crops and low-input tree/agroforestry systems. Plot, farm and regional scales (where applicable) will be addressed. The investigation will include new agricultural practices, e.g. soil conservation, crop rotation management, seed and crop mixtures and economic issues relating to the costs of indicator measurement and to benefits of biodiversity as perceived by different groups of the population. Stakeholders (farming communities, conservation NGOs, administrators) will be integrated at critical stages of the indicator selection process. A handbook with factsheets will be produced for validated indicators and a sampling design for biodiversity monitoring in organic and low-input farming systems across Europe.


Yakymenko I.L.,Ukrainian Academy of Sciences | Yakymenko I.L.,Bila Tserkva National Agrarian University | Sidorik E.P.,Ukrainian Academy of Sciences | Tsybulin O.S.,Bila Tserkva National Agrarian University
Ukrain'skyi Biokhimichnyi Zhurnal | Year: 2011

Review is devoted to the analysis of biological effects of microwaves. The results of last years' researches indicated the potential risks of long-term low-level microwaves exposure for human health. The analysis of metabolic changes in living cells under the exposure of microwaves from mobile communication systems indicates that this factor is stressful for cells. Among the reproducible effects of low-level microwave radiation are overexpression of heat shock proteins, an increase of reactive oxygen species level, an increase of intracellular Ca 2+, damage of DNA, inhibition of DNA reparation, and induction of apoptosis. Extracellular-signalregulated kinases ERK and stress-related kinases p38MAPK are involved in metabolic changes. Analysis of current data suggests that the concept of exceptionally thermal mechanism of biological effects of microwaves is not correct. In turn, this raises the question of the need to revaluation of modern electromagnetic standards based on thermal effects of non-ionizing radiation on biological systems.


Yakymenko I.,Bila Tserkva National Agrarian University | Yakymenko I.,R.E. Kavetsky Institute of Experimental Pathology | Sidorik E.,R.E. Kavetsky Institute of Experimental Pathology
Experimental Oncology | Year: 2010

Intensive implementation of mobile telephony technology in everyday human life during last two decades has given a possibility for epidemiological estimation of long-term effects of chronic exposure of human organism to low-intensive microwave (MW) radiation. Latest epidemiological data reveal a significant increase in risk of development of some types of tumors in chronic (over 10 years) users of mobile phone. It was detected a significant increase in incidence of brain tumors (glioma, acoustic neuroma, meningioma), parotid gland tumor, seminoma in long-term users of mobile phone, especially in cases of ipsilateral use (case-control odds ratios from 1.3 up to 6.1). Two epidemiological studies have indicated a significant increase of cancer incidence in people living close to the mobile telephony base station as compared with the population from distant area. These data raise a question of adequacy of modern safety limits of electromagnetic radiation (EMR) exposure for humans. For today the limits were based solely on the conception of thermal mechanism of biological effects of RF/MW radiation. Meantime the latest experimental data indicate the significant metabolic changes in living cell under the low-intensive (non-thermal) EMR exposure. Among reproducible biological effects of low-intensive MWs are reactive oxygen species overproduction, heat shock proteins expression, DNA damages, apoptosis. The lack of generally accepted mechanism of biological effects of low-intensive non-ionizing radiation doesn't permit to disregard the obvious epidemiological and experimental data of its biological activity. Practical steps must be done for reasonable limitation of excessive EMR exposure, along with the implementation of new safety limits of mobile telephony devices radiation, and new technological decisions, which would take out the source of radiation from human brain. Copyright © Experimental Oncology, 2010.


Yakymenko I.,R.E. Kavetsky Institute of Experimental Pathology | Tsybulin O.,Bila Tserkva National Agrarian University | Sidorik E.,R.E. Kavetsky Institute of Experimental Pathology | Henshel D.,Indiana University Bloomington | And 2 more authors.
Electromagnetic Biology and Medicine | Year: 2015

This review aims to cover experimental data on oxidative effects of low-intensity radiofrequency radiation (RFR) in living cells. Analysis of the currently available peer-reviewed scientific literature reveals molecular effects induced by low-intensity RFR in living cells; this includes significant activation of key pathways generating reactive oxygen species (ROS), activation of peroxidation, oxidative damage of DNA and changes in the activity of antioxidant enzymes. It indicates that among 100 currently available peer-reviewed studies dealing with oxidative effects of low-intensity RFR, in general, 93 confirmed that RFR induces oxidative effects in biological systems. A wide pathogenic potential of the induced ROS and their involvement in cell signaling pathways explains a range of biological/health effects of low-intensity RFR, which include both cancer and non-cancer pathologies. In conclusion, our analysis demonstrates that low-intensity RFR is an expressive oxidative agent for living cells with a high pathogenic potential and that the oxidative stress induced by RFR exposure should be recognized as one of the primary mechanisms of the biological activity of this kind of radiation. © 2015 Informa Healthcare USA, Inc. All rights reserved: reproduction in whole or part not permitted


Nepochatenko V.A.,Bila Tserkva National Agrarian University
Ferroelectrics | Year: 2010

The method of determination of functional dependence between parameters of crystalline lattice in ferroelastics and multiaxial ferroelectrics based on the condition of orientation matching of domains is offered. The analytical type of functional dependence has been found for phase transitions m3m-F4mm and 4/m-F2/m. We have compared theoretical results with experimental data for BaTiO3 and BiVO4. Copyright © Taylor & Francis Group, LLC.


Yakymenko I.,R.E. Kavetsky Institute of Experimental Pathology | Yakymenko I.,Bila Tserkva National Agrarian University | Sidorik E.,R.E. Kavetsky Institute of Experimental Pathology | Kyrylenko S.,Masaryk University | Chekhun V.,R.E. Kavetsky Institute of Experimental Pathology
Experimental Oncology | Year: 2011

In this review we discuss alarming epidemiological and experimental data on possible carcinogenic effects of long term exposure to low intensity microwave (MW) radiation. Recently, a number of reports revealed that under certain conditions the irradiation by low intensity MW can substantially induce cancer progression in humans and in animal models. The carcinogenic effect of MW irradiation is typically manifested after long term (up to 10 years and more) exposure. Nevertheless, even a year of operation of a powerful base transmitting station for mobile communication reportedly resulted in a dramatic increase of cancer incidence among population living nearby. In addition, model studies in rodents unveiled a significant increase in carcinogenesis after 17-24 months of MW exposure both in tumor-prone and intact animals. To that, such metabolic changes, as overproduction of reactive oxygen species, 8-hydroxi-2- deoxyguanosine formation, or ornithine decarboxylase activation under exposure to low intensity MW confirm a stress impact of this factor on living cells. We also address the issue of standards for assessment of biological effects of irradiation. It is now becoming increasingly evident that assessment of biological effects of non-ionizing radiation based on physical (thermal) approach used in recommendations of current regulatory bodies, including the International Commission on Non-Ionizing Radiation Protection (ICNIRP) Guidelines, requires urgent reevaluation. We conclude that recent data strongly point to the need for re-elaboration of the current safety limits for non-ionizing radiation using recently obtained knowledge. We also emphasize that the everyday exposure of both occupational and general public to MW radiation should be regulated based on a precautionary principles which imply maximum restriction of excessive exposure. Copyright © Experimental Oncology, 2011.


Tsybulin O.,R.E. Kavetsky Institute of Experimental Pathology | Tsybulin O.,Bila Tserkva National Agrarian University | Sidorik E.,R.E. Kavetsky Institute of Experimental Pathology | Brieieva O.,R.E. Kavetsky Institute of Experimental Pathology | And 5 more authors.
International Journal of Radiation Biology | Year: 2013

Purpose: Our study was designed to assess the effects of low intensity radiation of a GSM (Global System for Mobile communication) 900 MHz cellular phone on early embryogenesis in dependence on the duration of exposure. Materials and methods: Embryos of Japanese Quails were exposed in ovo to GSM 900 MHz cellular phone radiation during initial 38 h of brooding or alternatively during 158 h (120 h before brooding plus initial 38 h of brooding) discontinuously with 48 sec ON (average power density 0.25 μW/cm2, specific absorption rate 3 μW/kg) followed by 12 sec OFF intervals. A number of differentiated somites were assessed microscopically. Possible DNA damage evoked by irradiation was assessed by an alkaline comet assay. Results: Exposure to radiation from a GSM 900 MHz cellular phone led to a significantly altered number of differentiated somites. In embryos irradiated during 38 h the number of differentiated somites increased (p < 0.001), while in embryos irradiated during 158 h this number decreased (p < 0.05). The lower duration of exposure led to a significant (p < 0.001) decrease in a level of DNA strand breaks in cells of 38-h embryos, while the higher duration of exposure resulted in a significant (p < 0.001) increase in DNA damage as compared to the control. Conclusion: Effects of GSM 900 MHz cellular phone radiation on early embryogenesis can be either stimulating or deleterious depending on the duration of exposure. © 2013 Informa UK, Ltd.


Nepochatenko V.A.,Bila Tserkva National Agrarian University
Bulletin of the Russian Academy of Sciences: Physics | Year: 2010

A method for determining the functional dependence between lattice parameters from the condition of orientation correlation between domains in ferroelastics and multiaxial ferroelectrics is proposed. The obtained analytical dependences are compared with experimental data on BaTiO3 and PbTiO3 for the m3mF4mm phase transition. It is shown that the domain structure in PbTiO3 is strained. © 2010 Allerton Press, Inc.


Nepochatenko V.A.,Bila Tserkva National Agrarian University | Nepochatenko I.A.,Bila Tserkva National Agrarian University
Ferroelectrics | Year: 2016

Method of determining temperature dependence of lattice parameters and crystal cell volume of ferroelastics in analytical form at 4 / m F 2 / m structural phase transition has been suggested on the basis of conservation condition of paraelastic phase macrosymmetry in a polydomain crystal. The analysis of functions for the sample of BiVO4 was made. Temperature dependence of the spontaneous strain components and crystallographic axes spontaneous rotation are determined for the crystal. © 2016 Taylor & Francis Group, LLC.


PubMed | Bila Tserkva National Agrarian University, Ukrainian Academy of Sciences, University of Eastern Finland, University of Campinas and Indiana University Bloomington
Type: Journal Article | Journal: Electromagnetic biology and medicine | Year: 2016

This review aims to cover experimental data on oxidative effects of low-intensity radiofrequency radiation (RFR) in living cells. Analysis of the currently available peer-reviewed scientific literature reveals molecular effects induced by low-intensity RFR in living cells; this includes significant activation of key pathways generating reactive oxygen species (ROS), activation of peroxidation, oxidative damage of DNA and changes in the activity of antioxidant enzymes. It indicates that among 100 currently available peer-reviewed studies dealing with oxidative effects of low-intensity RFR, in general, 93 confirmed that RFR induces oxidative effects in biological systems. A wide pathogenic potential of the induced ROS and their involvement in cell signaling pathways explains a range of biological/health effects of low-intensity RFR, which include both cancer and non-cancer pathologies. In conclusion, our analysis demonstrates that low-intensity RFR is an expressive oxidative agent for living cells with a high pathogenic potential and that the oxidative stress induced by RFR exposure should be recognized as one of the primary mechanisms of the biological activity of this kind of radiation.

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