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Beijing, China

Beijing Sport University , founded on November 1, 1953, has contributed greatly to the sustainable development of Chinese sport and physical education and has enjoyed the fame as one of the key sport universities in China.In the past five decades, over 30,000 students of sports-related majors have graduated from BSU. There is a wide range of academic courses and majors studies, and the number of master and doctoral positions ranks the first among its counterparts in China.Currently, BSU consists of seven colleges, two elite sports schools and has a large number of qualified teachers, among whom 227 are professors and associate professors. Its excellent staff and facilities can satisfy the needs of teaching, training and scientific research. BSU has established friendly cooperative relations and intercollegiate relations with 28 universities and tens of non-governmental organizations in 16 countries.BSU was instrumental in the popularization of skateboarding in China. Wikipedia.

Xiao C.-M.,Beijing Institute of Graphic Communication | Zhuang Y.-C.,Beijing Sport University
Journal of the American Geriatrics Society | Year: 2015

Objectives To investigate the effectiveness of a 6-month Liuzijue qigong (LQG) program in promoting physical and psychosocial function in individuals with chronic obstructive pulmonary disease (COPD). Design Single-blind, randomized controlled trial. Setting Seven hospital respiratory care centers in Beijing. Participants Individuals with COPD (N = 126; mean age 71.1 ± 2.7, range 65-85). Methods Participants were randomly assigned to one of two groups: LQG (n = 63) and control (n = 63). Subjects in the LQG group received a LQG program consisting of four 45-minute sessions each week and daily walking for 30 minutes for 6 months. Control subjects walked daily for 30 minutes. Data collection was performed at baseline, at 6 weeks, and at 6 months. Primary outcomes involved functional capacity, and secondary outcomes involved quality of life. Results The LQG group had greater improvements in the 6-minute walk test (P =.02); specific airway conductance (P =.02); monitored functional task evaluation (P =.04); Medical Outcomes Study 36-item Short-Form Health Survey (SF-36; general health (P <.001), mental health (P =.03)); Chronic Respiratory Questionnaire (dyspnea (P =.05), emotion (P =.05), and mastery (P =.04) at 6-month follow-up. After 6 months, the control group had significant improvement only on the SF-36 mental health (P =.02). Conclusion LQG promoted functional capacity and quality of life in older adults with COPD at 6 months and is a good alternative home exercise program for older adults in the rehabilitation of COPD. © 2015 The American Geriatrics Society.

Zhao S.-T.,Guangzhou University | Zhao L.,Beijing Sport University | Li J.-H.,Guangzhou University
Neurochemical Research | Year: 2013

Humanin (HN) has been proved to be an extensive neuroprotective peptide against AD-related and unrelated insults, but little is know about the effect of HN in inflammation response. Current studies indicated the receptors of HN have a close relationship with immune system, which led us to hypothesize HN might have a role in inflammatory response. In this study, we used lipopolysaccharide (LPS) to induce astrocyte inflammation response. This model in vitro allowed us to study the effect of HN on the pure response of astrocyte without the exogenous influence between cells in vivo. Our results showed that 1.0 μg/ml LPS induced a significant activation of astrocyte, shown as the marked increase in the glial fibrillary acidic protein (GFAP) expression, the cell viability and the number of 5-bromo-2′-deoxyuridine (BrdU)-positive living cells. Pretreatment with HN (5, 10, 20 μM) led to a significant inhibition in astrocyte overactivation in a concentration dependent manner. We also found pretreatment with HN decreased the level of proinflammatory cytokines, interleukin (IL)-6, IL-1β and tumor necrosis factor α (TNFα) induced by LPS. Furthermore, we noticed HN couldn't completely reverse the above inflammatory injury. Our findings imply that HN partly antagonizes inflammation injury induced by LPS and the protective effect of HN on astrocyte is concentration-dependent. © 2012 Springer Science+Business Media New York.

In this paper, we investigate a generalized inhomogeneous fifth-order nonlinear Schrödinger equation, generated by deforming the inhomogeneous Heisenberg ferromagnetic spin system through the space curve formalism. Based on the Ablowitz-Kaup-Newell-Segur system, infinitely many conservation laws will be obtained. Via the introduction of the auxiliary functions, bilinear form and N-soliton solutions have been derived with symbolic computation. Propagation and interaction of solitons have been studied through the analytical results. Effects of the inhomogeneous functions f = μ 1 x + ν 1 and h = μ 2 x + ν 2 on the soliton velocity and interactions have been discussed graphically and analytically. © 2014 EDP Sciences, SIF, Springer-Verlag Berlin Heidelberg.

Kong Z.,University of Macau | Zang Y.,University of Macau | Hu Y.,Beijing Sport University
Sleep and Breathing | Year: 2013

Background: Intermittent normobaric hypoxia training, an alternative to altitude training for athletes, may be beneficial to treat overweight and obesity. The purpose of this study is to investigate whether normobaric hypoxia training combined with low-caloric diet has the additive effect on weight loss compared with normoxia training in obese young adults. Methods: Twenty-two subjects (age 17-25 years, body mass index >27.5 kg/m2) were recruited for a 4-week residential camp of weight loss with low caloric intake, and trained at 60-70 % maximal heart rate of aerobics and 40-50 % of maximal strength of training. They were randomly assigned to either a normobaric hypoxia (HT, FiO2 = 16.4-14.5 %) or normoxia training group (NT, FiO2 = 21 %), and subjects in HT and NT groups experienced weekly 16-h normoxia and 6-h hypoxia or 22-h normoxia training, respectively. Body composition, resting blood pressure (BP) and brachial-ankle pulse wave velocity (baPWV) were determined before and after the intervention. Results: Weight loss was found in HT (-6.9 kg or -7.0 %, p < 0.01) and NT groups (-4.3 kg or -4.2 %, p < 0.01) significantly, and the former lost more weight than the latter (p < 0.01). Hypoxia training improved systolic BP (-7.6 %) and mean BP (-7.1 %) significantly (p < 0.05) despite having no effect on baPWV. Conclusion: Four weeks of normobaric hypoxia residential training with low caloric diet has an additive improvement on weight loss. It seems that normobaric hypoxia training might be a promising method to treat obesity. © 2013 Springer-Verlag Berlin Heidelberg.

Shi L.,Beijing Sport University
Arteriosclerosis, Thrombosis, and Vascular Biology | Year: 2016

OBJECTIVE—: Regular exercise is an effective nonpharmacological means of preventing and controlling hypertension. However, the molecular mechanisms underlying its effects remain undetermined. The hypothesis that hypertension increases the functional coupling of large-conductance Ca-activated K (BKCa) channels with ryanodine receptors in spontaneously hypertensive rats (SHR) as a compensatory response to an increase in intracellular Ca concentration in cerebral artery smooth muscle cells was assessed here. It was further hypothesized that exercise training would prevent this increase in functional coupling. APPROACH AND RESULTS—: SHR and Wistar–Kyoto (WKY) rats were randomly assigned to sedentary groups (SHR-SED and WKY-SED) and exercise training groups (SHR-EX and WKY-EX). Cerebral artery smooth muscle cells displayed spontaneous transient outward currents at membrane potentials more positive than −40 mV. The amplitude of spontaneous transient outward currents together with the spontaneous Ca sparks in isolated cerebral artery smooth muscle cells was significantly higher in SHR-SED than in WKY-SED. Moreover, hypertension displayed increased whole-cell BKCa, voltage-gated Ca channel, but decreased KV currents in cerebral artery smooth muscle cells. In SHRs, the activity of the single BKCa channel increased markedly, and the protein expression of BKCa (β1, but not α-subunit) also increased, but KV1.2 decreased significantly. Exercise training ameliorated all of these functional and molecular alterations in hypertensive rats. CONCLUSIONS—: These data indicate that hypertension leads to enhanced functional coupling of ryanodine receptors–BKCa to buffer pressure–induced constriction of cerebral arteries, which attributes not only to an upregulation of BKCa β1-subunit function but also to an increase of Ca release from ryanodine receptors. However, regular aerobic exercise efficiently prevents augmented coupling and so alleviates the pathological compensation and restores cerebral arterial function. © 2016 American Heart Association, Inc.

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